A Drosophila model of GDAP1 function reveals the involvement of insulin signalling in the mitochondria-dependent neuromuscular degeneration.

Por: López Del Amo V, Palomino-Schätzlein M, Seco-Cervera M, García-Giménez JL, Pallardó FV, Pineda-Lucena A, Galindo MI

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Publicada: 1 mar 2017

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Filiaciones:
López Del Amo V:
 Centro de Investigación Príncipe Felipe, 46012 Valencia, Spain

 Center for Biomedical Network Research on Rare Diseases (CIBERER), 46012 Valencia, Spain

Palomino-Schätzlein M:
 Centro de Investigación Príncipe Felipe, 46012 Valencia, Spain

Seco-Cervera M:
 Center for Biomedical Network Research on Rare Diseases (CIBERER), 46012 Valencia, Spain

 Department of Physiology, School of Medicine and Dentistry, Universitat de València, INCLIVA Biomedical Research Institute, 46010 Valencia, Spain

García-Giménez JL:
 Center for Biomedical Network Research on Rare Diseases (CIBERER), 46012 Valencia, Spain

 Department of Physiology, School of Medicine and Dentistry, Universitat de València, INCLIVA Biomedical Research Institute, 46010 Valencia, Spain

Pallardó FV:
 Center for Biomedical Network Research on Rare Diseases (CIBERER), 46012 Valencia, Spain

 Department of Physiology, School of Medicine and Dentistry, Universitat de València, INCLIVA Biomedical Research Institute, 46010 Valencia, Spain

Pineda-Lucena A:
 Centro de Investigación Príncipe Felipe, 46012 Valencia, Spain

 Drug Discovery Unit, Instituto de Investigación Sanitaria La Fe, 46026 Valencia, Spain

Galindo MI:
 Centro de Investigación Príncipe Felipe, 46012 Valencia, Spain

 IDM-Institute of Molecular Recognition, Universidad Politécnica de Valencia, 46022 Valencia, Spain

 UPV-CIPF Joint Unit Disease Mechanisms and Nanomedicine, Valencia, Spain